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Autoregulation of ccl26 synthesis and secretion in a549 cells: a possible mechanism by which alveolar epithelial cells modulate airway inflammation
(2005)
Abonyo, B. O., M. S. Alexander, and A. S. Heiman. Autoregulation of CCL26 synthesis and secretion in A549 cells: a possible
mechanism by which alveolar epithelial cells modulate airway inflammation. Am J Physiol Lung Cell ...
Characterization of -soluble n-ethylmaleimide–sensitive fusion attachment protein in alveolar type ii cells implications in lung surfactant secretion
(2002)
N-ethylmaleimide–sensitive fusion protein (NSF) and soluble NSF attachment protein (α-SNAP) are thought to be soluble factors that transiently bind and disassemble SNAP receptor complex during exocytosis in neuronal and ...
CCL26-targeted siRNA treatment of alveolar type II cells decreases expression of CCR3-binding chemokines and reduces eosinophil migration: implications in asthma therapy
(2009)
The underlying infl ammation present in chronic airway diseases is orchestrated by increased expression of CC
chemokines that selectively recruit leukocyte populations into the pulmonary system. Human CCL26 signals
through ...
Cytokine-stimulated human lung alveolar epithelial cells release eotaxin-2 (CCL24) and eotaxin-3 (CCL26)
(Mary Ann Liebert, Inc, 2005)
Asthma is a complex inflammatory disease characterized by a prolonged underlying airway inflammation resulting from cytokine-orchestrated signaling between many types of cells, including airway epithelial cells.
Trafficking, ...
Reorganization of cytoskeleton during surfactant secretion in lung type II cells: a role of annexin II
(Elsevier, 2003)
The secretion of lung surfactant requires the movement of lamellar bodies to the plasma membrane through cytoskeletal barrier at the
cell cortex. We hypothesized that the cortical cytoskeleton undergoes a transient ...
Post-transcriptional silencing of CCR3 downregulates IL-4 stimulated release of eotaxin-3 (CCL26) and other CCR3 ligands in alveolar type II cells
(2008)
Trafficking and inflammation in airway diseases are, in part, modulated by members of the CC chemokine family, eotaxin-1 (CCL11), eotaxin-2 (CCL24), and eotaxin-3 (CCL26), which transduce signals through their CCR3 receptor. ...
Syntaxin 2 and SNAP-23 are required for regulated surfactant secretion
(2014)
The secretion of lung surfactant in alveolar type II cells is a complex process involving the fusion of lamellar bodies with the plasma membrane. This process is somewhat different from the exocytosis of hormones and ...